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Once just about everyone has accepted that insulin is a satiety hormone it becomes properly obvious that individuals on low carbohydrate diets, using their chronically reduced insulin levels, should be hungry. After all, I have seen it suggested by Stephan that insulin may assist weight loss. I’m still trying to get my head around that one, while eating low carb and trying to remember what it felt prefer to be hungry.
Of course, as Stephan points out, hypoglycaemia is a powerful appetite stimulant. Again LC eaters, using their chronically low blood sugar levels, should be ravenous. I’m also trying to get my head round that one too. Reality sometimes gets in the way of great theories. I’m not quite sure where you can devote the neuronal insulin receptor knock-out mouse.
It’s fat, so the conclusion appears to be that brain insulin receptors are important to satiety. I’m sure they are. However, these extra fat mice are also hyperinsulinaemic and you will be lipolytically challenged. They do not have me mainlining insulin as a weight loss drug. I love the impaired spermatogenesis and ovarian follicular maturation too. I still wouldn’t normally decry leptin here but these hyperleptinaemic mice don’t do reproduction terribly well. Dare the I-word can be used by me when talking about fertility?
Insulin inhibits lipolysis. Don’t forget that whenever we come to discuss the Pima. Before we proceed let’s go through the satiating effects of foods. Stephan’s refs 4, 5, 6 and 7 suggest no macronutrient issues ref and much 8 shows protein is more satiating than carbohydrate. But reference 9 9 is the absolute beauty.